Neurogenic Claudication

Overview

Neurogenic claudication is a symptom caused by central lumbar stenosis. Most of the time, the stenosis is at L4-5, L3-4, or L2-3 with a combination of 1 or 2 continuous levels. Most patients present with stenosis at L4-5 alone or L3-4 & L4-5. Symptoms usually start as generalized back pain across the lower back made worse with standing, walking, and extension. Over time, the patient or family member may notice that the patient is frequently “hunched” or flexed forward a few minutes after they stand from a seated position and begin to walk. As they start walking, the pain worsens while flexion begins to relieve it.  When you see someone walking severely hunched over, they most likely have Neurogenic Claudication.  Pain then begins to travel down the legs, bilaterally, sometimes affecting the anterior (front) leg, posterior (back) leg, or both.  Once the patient sits down, symptoms begin to subside.  Stenosis is a component of Degenerative Disc Disease and can include central disc herniation and /or facet hypertrophy.  Enlargement or hypertrophy of the facet joint can be very painful, adding to the back pain. Compression of the nerves from lumbar stenosis causes leg pain or neurogenic claudication.  Once the symptoms start, they usually worsen over time.

A Spondylolisthesis at L5-S1 is called an “Isthmic” Spondylolisthesis and affects approximately 5 to 7% of the population. A Spondylolisthesis at L4-5 is a called a “Degenerative” Spondylolisthesis. Both types can be painful and present with back pain, leg pain or both.

If a patient develops a foot drop or other leg weakness, conservative care will be initiated but if the weakness persists, you may surgeon may need to strongly consider surgical decompression.

Spinal Stenosis (Cervical)

Treatment

Non-operative. Common treatments include non-steroidal anti-inflammatory agents, physical therapy, and epidural injections. Acupuncture and Chiropractic care can help as well. Patients are also candidates for facet blocks to alleviate back pain. There is a small nerve on the facet joint that does not participate in motor or sensory function of the leg; only sensation to facet joint and if injected, can relieve back pain associated with degeneration. Patients can also find relief from an epidural injection in the region of the stenosis. If successful, Physiatrists may inject up to three times in a 12-month period.

Surgery. Surgery may involve a simple decompression called a laminectomy or require a laminectomy with fusion.

FAQs

What is neurogenic claudication?

Neurogenic claudication is a condition characterized by pain, numbness, or weakness in the legs and buttocks. It occurs when the spinal nerves are compressed—most commonly due to lumbar spinal stenosis—leading to discomfort during activities such as walking or standing that typically improves with rest or leaning forward.

What causes neurogenic claudication?

The most common cause of neurogenic claudication is lumbar spinal stenosis, a narrowing of the spinal canal often due to age-related degenerative changes like disc degeneration, thickening of ligaments, and the formation of bone spurs. These changes reduce the space available for the spinal nerves, resulting in compression and the associated symptoms.

What symptoms are associated with neurogenic claudication?

Patients may experience a range of symptoms including:

  • Leg pain, cramping, or aching that worsens with prolonged walking or standing
  • Numbness or tingling in the legs or buttocks
  • Weakness in the lower extremities
  • Symptom relief when bending forward, sitting, or resting

These symptoms are typically activity-dependent and improve with positions that relieve pressure on the nerves.

How is neurogenic claudication diagnosed and treated?

Diagnosis begins with a thorough medical history and physical examination, with attention to how symptoms change with different activities or positions. Imaging studies, such as MRI or CT scans, are used to evaluate the extent of spinal canal narrowing and nerve compression. Treatment generally starts with conservative approaches like physical therapy, pain management, and activity modification. If symptoms persist or worsen, more advanced interventions—such as corticosteroid injections or surgical decompression—may be recommended to relieve nerve compression and improve quality of life.

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